peptide wiki

SS-31

aka Elamipretide

Growth Hormone Secretagogues

Mechanism

SS-31 (elamipretide) is a mitochondria-targeted tetrapeptide (D-Arg-Dmt-Lys-Phe-NH2) developed by Hazel Szeto (Cornell) and clinically advanced by Stealth BioTherapeutics. It concentrates in the inner mitochondrial membrane by binding cardiolipin. In September 2025, the FDA granted accelerated approval to elamipretide (brand name FORZINITY) as the first therapy for Barth syndrome — the first FDA-approved mitochondria-targeted drug. It remains investigational for other indications.

Identification

CAS number736992-21-5
Molecular formulaC32H49N9O5
SequenceD-Arg-Dmt-Lys-Phe-NH2
Typical dose40 mg/day subcutaneous (clinical); research range 0.1–2 mg/kg/day subcutaneous
Half-life~4 hours (subcutaneous)

Vendors selling SS-31

VendorSizePriceListed purityTrust
Peptide Partners 10 mg $110.00 99
Peptide Partners 50 mg $320.00 99
Moglabs 10 mg $46.00 96
Orbitrex Peptides $79.99 95
Profound Aminos 10 mg $49.00 95
Reta One Labs 10 mg $34.00 95
Licensed Peptides 10 mg $99.99 95
Licensed Peptides 50 mg $99.99 95
Mile High Compounds $69.99 94
Hydro Research Peptides 50 mg $1.75 91
Peptide Plugs 10 mg $40.00 89
Peptide Plugs 50 mg $130.00 89
Peptidegurus 10 mg 84
Peptidegurus 10 mg 84
Peptidegurus 25 mg 84
Peptidegurus 50 mg 84
Ascension Peptides 10 mg $60.00 84
Reta Peptide 83
Cocer Peptides 10 mg $120.00 82
OROS Research 10 mg 79
OROS Research 30 mg 79
Texpeptide 10 mg $55.00 70
Crystal Peptides 10 mg $49.99 66
Qing Li Peptide 63
Qing Li Peptide 10 mg 63
Peptiatlas 51
Taishijie 51
Taikangbio 50
MEI-PEPETIDE 10 mg $85.00 48
DongCheng Peptide 46
Mandy 10 mg $74.00 43
HongDa Peptide $109.00 43
zztai Peptide Ltd 43
MJT peptides 10 mg $106.00 39
Alpha Peptides
Strate Labs 50 mg $144.95
Atomik Labz 50 mg $199.00
Bulk Peptides 10 mg $200.00
Genetic Peptide 10 mg $60.00
Genetic Peptide 50 mg $60.00
Nextech Labs 50 mg $115.00
Peptira 50 mg $139.00
Peptira 10 mg $49.00
Pepvida Labs 50 mg $159.00
Platinum Lion 50 mg $139.99
Polaris Peptides 25 mg $129.00
Solution Peptides 50 mg $199.00
Soma Peptides 10 mg $59.00
Soma Peptides 25 mg $59.00
True Peptide 50 mg $160.00
True Peptide 10 mg $50.00
True Peptide 50 mg $50.00
Wellness Peptides 10 mg $55.00
Wellness Peptides 50 mg $55.00

Research

Mitochondrial-targeted SS-31 peptide attenuates radiation-induced cardiomyocyte senescence
Xie L, Wu J, Fan J, Krager KJ, Aykin-Burns N, Li S, Børsheim E, Qi X, Boerma M, Zhang H. · Journal of radiation research (2026) ·

Exposure to ionizing radiation, such as from radiation therapy or accidental radiation exposure can have adverse effects on the heart. While radiation injury in the heart may include cardiomyocyte senescence, there are no available strategies to prevent this phenomenon.

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Mitochondrial-Targeted SS-31 Attenuates the Doxorubicin-Induced Cardiomyoblast H9C2 Cell Senescence
Fan J, Wu J, Yan S, Li S, Rabinovitch PS, Qi X, Zhang H. · Biology (2026) · in-vitro

Doxorubicin (DOX), an effective chemotherapeutic agent for many types of cancer, is known for significant cardiotoxic side effects, which largely limit its clinical usage.

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Targeting Mitochondrial Dysfunction With Elamipretide (SS-31) Improves Skeletal Muscle Performance in a HFpEF Rat Model
Vahle B, Weidner S, Tomalka A, Schauer A, Augstein A, Männel A, Barthel P, Friedrich J, Beck G, Labeit S, Bowen TS, Siebert T, Linke A, Adams V. · Circulation. Heart failure (2026) · animal

Background Exercise intolerance, promoted by skeletal muscle- and mitochondrial dysfunction, has been identified as a therapeutic target in heart failure with preserved ejection fraction (HFpEF).

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Therapeutic Peptide SS-31 Modulates Membrane Binding and Aggregation of α-Synuclein and Restores Impaired Mitochondrial Function
Stefaniak E, Cui B, Yan X, Sun K, Teng X, Ying L. · Chemical biology & drug design (2026) ·

Membrane binding and aggregation properties of α-synuclein are closely associated with Parkinson's disease and a class of related syndromes named as synucleinopathy.

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Elamipretide (SS-31) and Nicotinamide Mononucleotide (NMN) Combination Therapy Targets TREM2 to Mitigate Post-ischemic Brain Injury in Mice
Yang Q, Zhang Y, Liu P, Yan Z, Li C, Tang Y, Yang Q. · Neurochemical research (2026) · animal

Ischemic stroke is a severe cerebrovascular disorder characterized by a cascade of pathological processes, including neuroinflammation and apoptosis. These processes lead to high mortality rates and long-term disabilities, imposing substantial socioeconomic burdens.

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Elamipretide (SS-31) promotes recovery by preserving mitochondrial bioenergetics and neural remodeling after spinal cord injury
Song Z, Ban Z, Zhao H, Mei X. · Neurochemistry international (2026) · animal

Spinal cord injury (SCI) induces secondary damage characterized by mitochondrial dysfunction, oxidative stress, and apoptosis, which collectively impede neurological recovery. Elamipretide (SS-31) is a mitochondria-targeting peptide with potential neuroprotective effects.

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SS-31 improves the quality of maternally aged oocytes by ameliorating mitochondrial function and metabolism
Xiong D, Zhang Y, Wei J, Wang L, Zhang G, Yu L, Zeng J, Liu W. · Journal of ovarian research (2026) · animal

Reproductive aging is closely associated with poor oocyte quality in vitro maturation, but effective approaches to ameliorate it have still not been fully determined. Here, we found that SS-31 supplementation efficaciously improved oocyte maturation and early embryonic development from aged mice.

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Mechanisms of Anti-Oxidants, N-Acetylcysteine and Elamipretide (SS-31), on Ozone-Induced Airway Hyperresponsiveness and Mucus Hypersecretion
Xie M, Weng J, Li C, Liu Q, Feng Y, Zhang H, Chang Q, Chung KF, Adcock IM, Li F, Fan X. · Lung (2026) ·

Background Ozone (O₃) exposure induces acute airway injury characterized by airway hyperresponsiveness (AHR) and airway mucus hypersecretion (AMH). Oxidative stress and mitochondria-derived reactive oxygen species (mtROS) are key contributors.

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SS-31 improves post-cardiac arrest brain injury by inhibiting microglial ferroptosis and polarization
Jiang T, Zhang H, Sun Y, Ji X, Xue L, Pan C, Guo Y, Xu F. · Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics (2026) · animal

Accumulating evidence suggests that ferroptosis and mitochondrial dysfunction contribute significantly to brain injury following cardiac arrest (CA) and resuscitation.

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Endothelial Glycocalyx Degradation as a Mediator of Neuroinflammation and Cognitive Impairment in Aged Rats: Protective Role of SS-31
Kan MH, Liu Y, Meng FQ, Fan L, Wu Y, Wang TL. · Molecular neurobiology (2025) · animal

To investigate the role of endothelial glycocalyx (eGC) degradation in mediating the progression from systemic inflammation to neuroinflammation and cognitive impairment in aged rats, and to explore the protective effects of the mitochondrial-targeted peptide SS-31.

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SS-31 Targets NOS2 to Enhance Osteogenic Differentiation in Aged BMSCs by Restoring Mitochondrial Function
Duan S, Zhang Q, Zhu J, Wang J. · Organogenesis (2025) ·

This study delves into the rejuvenating effects of SS-31 on aged human Bone Marrow-Derived Mesenchymal Stem Cells (BM-MSCs), focusing on its potential to restore their diminished osteogenic differentiation capacity, a critical issue in geriatric medicine and bone tissue engineering.

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SS-31: A promising therapeutic agent against bleomycin-induced pulmonary fibrosis in Mice
Gu Q, Wang Y, Zhang H, Yang W, Meng X, Zhao M. · PloS one (2025) · animal

Objective The aim of this research was to investigate if the mitochondria- targeting peptide SS-31 could serve as a protective measure against bleomycin-induced pulmonary fibrosis in mice. Method Mice were split into four groups named CON group, SS-31 group, BLM group, and the BLM + SS-31 group.

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FRDA Investigator Initiated Study (IIS) With Elamipretide
· ClinicalTrials.gov (2022) · rct

To evaluate the safety, tolerability, and activity of Elamipretide in treating vision loss in Friedreich Ataxia (FRDA).

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Evaluation of the Regeneration Capacity of Satellite Cells From the Quadriceps Compared to That of the Diaphragm
· ClinicalTrials.gov (2022) · rct

Actually no treatment exists to prevent the loss of diaphragmatic function induced by mechanical ventilation during an intensive care unit stay. The consequence is a growing number of survivors with moderate to severe chronic respiratory disease so called Ventilation-Induced Diaphragmatic Dysfunction (VIDD).

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